By Hans G. Beger, Akimasa Nakao, John P. Neoptolemos, Shu You Peng, Michael G. Sarr
ISBN-10: 0470673184
ISBN-13: 9780470673188
Pancreatic melanoma, Cystic Neoplasms and Endocrine Tumors: prognosis and Management is a latest, expertly crafted and clinically concentrated consultant to the analysis, administration and best-practice care of sufferers struggling with pancreatic melanoma, cystic neoplasms and endocrine tumours.
Packed with notable figures and as regards to the prime society directions, its major concentration is at the many endoscopic and radiologic diagnostic suggestions, clinical and surgical administration of either full-blown melanoma and different tumors, and the dangers of every type of treatment. additionally lined intimately are problems with tumor recurrence and long term consequence of treatment.
Brought to you by means of hugely expert nationwide and overseas leaders within the uniqueness and an skilled editor group, this is often a useful consultant to practising gastroenterologists and surgeons within the clinic and medical setting, in addition to oncologists and endocrinologists handling sufferers with pancreatic tumorous lesions.
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Extra info for Pancreatic Cancer, Cystic Neoplasms and Endocrine Tumors: Diagnosis and Management
Example text
TX, local invasion cannot be assessed; N0, no evidence of lymph node metastasis; N1, metastasis to Group 1 lymph nodes alone; N2, metastasis to Group 2 lymph nodes; N3, metastasis to Group 3 lymph nodes; NX, lymph node metastasis cannot be assessed; M0, no distant metastasis; M1, distant metastasis present; MX, distant metastasis cannot be assessed. References 1 Yanagisawa A, Kato Y. Development of pancreatic cancer. The Japanese Society of Gastroenterology. 2004;101: 1061–1071. tumor limited to the pancreas, 2 cm or less in greatest dimension; T2, tumor limited to the pancreas, more than 2 cm in greatest dimension; T3, tumor extends beyond pancreas, but without involvement of celiac axis or superior mesenteric artery; T4, tumor involves celiac axis or superior mesenteric artery; TX, primary tumor cannot be assessed; N0, no regional lymph node metastasis; N1, regional lymph node metastasis; NX, regional lymph nodes cannot be assessed; M0, no distant metastasis; M1, distant metastasis.
The most extensively applied tumor-associated marker is carbohydrate antigen (CA) 19-9. Although CA19-9 was initially considered useful in the management of patients with colorectal carcinoma, its role in pancreatic cancer has become more evident (7). The reported sensitivity and specificity rates of CA19-9 for pancreatic cancer range from 70% to 92% and from 68% to 92%, respectively (8,9). However, sensitivity is closely associated with tumor size. Levels of CA19-9 are of limited value for diagnosing small tumors (10,11).
2012;3:339–347. 55 Bussom S, Saif MW. Methods and rationale for the early detection of pancreatic cancer. Highlights from the “2010 ASCO Gastrointestinal Cancers Symposium”. JOP: Journal of the Pancreas. 2010;11:128–130. 56 Brentnall TA, Bronner MP, Byrd DR, et al. Early diagnosis and treatment of pancreatic dysplasia in patients with a family history of pancreatic cancer. Annals of Internal Medicine. 1999;131:247–255. 57 Kimmey MB, Bronner MP, Byrd DR, et al. Screening and surveillance for hereditary pancreatic cancer.
Pancreatic Cancer, Cystic Neoplasms and Endocrine Tumors: Diagnosis and Management by Hans G. Beger, Akimasa Nakao, John P. Neoptolemos, Shu You Peng, Michael G. Sarr
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