Download Pediatric gastrointestinal disease : pathophysiology, by W Allan Walker; et al PDF

By W Allan Walker; et al

ISBN-10: 0815190824

ISBN-13: 9780815190820

This textbook comprises sections on improvement, pathophysiology, scientific manifestations and administration, and data had to diagnose and deal with sufferers. It additionally appears on the ideas of treatment, hence delivering details at the administration of gastrointestinal ailment states, together with rehydration and nutrients, drugs, biopsychosocial and surgical aid. This variation comprises the newest advancements, corresponding to gene remedy, hepatitis, AIDS, and transplantation

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Additional info for Pediatric gastrointestinal disease : pathophysiology, diagnosis, management

Example text

For instance, TGF-β1 selectively augments collagen synthesis by intestinal smooth muscle cells,236 a response that contributes to intestinal fibrosis. 124,125 CELL ADHESION MOLECULES The involvement of cell adhesion molecules in inflammation depends on the organ affected and the nature of the inflammatory stimulus. 94,243–246,249 Addi- CELL ADHESION MOLECULES CELL ADHESION MOLECULE MAIN CELLULAR SOURCE MAIN LIGAND MAIN TARGET SELECTIN FAMILY E-selectin L-selectin P-selectin Endothelial cells Lymphocytes, neutrophils, monocytes Platelets, endothelial cells L-selectin MAd-CAM-1 L-selectin Neutrophils, monocytes, T cells Mucosal HEV, endothelial cells Neutrophils, monocytes, platelets INTEGRIN FAMILY LFA-1 (CD11a/CD18) LFA-3 Mac-1 (CD11b/CD18) VLA-1, -2, -3 VLA-4 All leukocytes All cells, some T cells Neutrophils, monocytes Collagen, laminin Lymphocytes, monocytes ICAM-1, -2, -3 CD2 ICAM-1 and -3, fibrinogen ECM VCAM-1, fibronectin Lymphoid, nonlymphoid cells T cells, natural killer cells Lymphoid, nonlymphoid cells, ECM VLA-5 VLA-6 α4β7 Lymphocytes, monocytes Lymphocytes, monocytes Lymphocytes Fibronectin Laminin MAd-CAM-1, fibronectin Endothelial cells, fibroblasts, monocytes, ECM ECM ECM Mucosal HEV, ECM IMMUNOGLOBULIN SUPERFAMILY ICAM-1 (CD54) VCAM-1 (CD106) MAd-CAM-1 PECAM-1 Lymphoid, nonlymphoid cells Endothelial cells, fibroblasts, monocytes Mucosal HEV Endothelial cells, neutrophils LFA-1, Mac-1 VLA-4, α4β7 α4β7, L-selectin PECAM-1 Neutrophils, lymphocytes, monocytes Neutrophils, lymphocytes, monocytes Lymphocytes, monocytes, neutrophils Endothelial cells, neutrophils CADHERIN FAMILY Cadherin, α- and β-catenin Adjacent cells Cadherin Same cell type OTHER CD44 (Hermes) Lymphoid, nonlymphoid cells Hyaluronan, collagen Endothelial cells, ECM ECM = extracellular matrix; HEV = high endothelial venules; ICAM = intercellular cell adhesion molecule; LFA = leukocyte function–associated antigen; MAd-CAM = mucosal addressin cell adhesion molecule; PECAM = platelet–endothelial cell adhesion molecule; VCAM = vascular cell adhesion molecule; VLA = very late activation antigen.

Chapter 3 • Inflammation Mucosal Microvascular Endothelial Cells CONCLUSIONS FIGURE 3-1 Major cell adhesion molecules involved in the various steps (rolling, adhesion, and transmigration) necessary to move leukocytes from the intravascular to the interstitial space. Molecules expressed by leukocytes are listed to their left, and molecules expressed by endothelial cells are listed below them. ICAM = intracellular adhesion molecule; MAd-CAM = mucosal addressin cell adhesion molecule; PECAM = platelet–endothelial cell adhesion molecule; VCAM = vascular cell adhesion molecule; VLA = very late activation antigen.

Each portion of the gastrointestinal tract displays specialized features that reflect adaptation to particular physiologic and metabolic requirements so that the outcome of an inflammatory process may vary in different segments of the intestine.

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Pediatric gastrointestinal disease : pathophysiology, diagnosis, management by W Allan Walker; et al


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