By Gerald Rimbach; Jürgen Fuchs; Lester Packer
ISBN-10: 0824726634
ISBN-13: 9780824726638
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Lung disease: Ozone is a powerful oxidant associated with impairment of pulmonary function and increased airway inflammation. Romieu et al. studied 158 asthmatic children in Mexico city, an area with high ozone exposure and looked at the effect of GSTM1 genotype and antioxidant supplementation (vitamins C and E). The GSTM1-null genotype was present in 39% of the 22 Gill-Garrison, Slater, and Grimaldi children. In those given placebo, the deleterious effect of ozone on forced expiratory flow was worse in GSTM1-nulls than in those carrying the gene.
Corradi M, Alinovi R, Goldoni M, Vettori M, Folesani G, Mozzoni P, Cavazzini S, Bergamaschi E, Rossi L, Mutti A. Biomarkers of oxidative stress after controlled human exposure to ozone. Toxicol Lett 2002; 134(1 – 3):219– 225. 58. Rabinowitz PM, Pierce Wise J Sr, Hur Mobo B, Antonucci PG, Powell C, Slade M. Antioxidant status and hearing function in noise-exposed workers. Hear Res 2002; 173(1 – 2):164– 171. 59. Wilson MH, Grant PJ, Hardie LJ, Wild CP. Glutathione S-transferase M1 null genotype is associated with a decreased risk of myocardial infarction.
31) reported that $2% of the plasma donors in Sweden had an 8– 10-fold higher SOD3 level and that a single base substitution of C to G at position 760 of the cDNA was responsible for the high level in plasma. The polymorphism is located in the region associated with the heparin affinity of the enzyme. The resulting amino acid substitution may result in a decrease of heparin affinity which favors the presence of SOD3 in the serum; thus, the high plasma activity can be explained by an accelerated release from the tissue interstitium heparan sulfate to the vasculature which is accompanied by significantly reduced tissue SOD3 activities.
Nutrigenomics by Gerald Rimbach; Jürgen Fuchs; Lester Packer
by George
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